Who is
Majid Ali, M.D.
► Dr. Ali's CV

Majid Ali is a pioneer who is changing the face of medicine with his innovative and spirited approach.

His credentials are impeccable Complementary Medicine Journal

"I stand in awe of Ali's superb scientific knowledge, his insights into the nature of the the healing process and his ability to explain hard science."

Aubrey Worrell, MD
Past President, the American Academy of
Environmental Medicine

Majid Ali, M.D.
Editor, The Journal of Integrative Medicine

Formerly, Associate Professor of Pathology (adj.), College of Physicians and Surgeons of Columbia University, NY

Formerly, President of Staff and
Chief Pathologist, Holy Name Hospital, Teaneck, NJ

Fellow, Royal College of Surgeons of England - Diplomate,

American Board of Anatomic and Clinical Pathology

Diplomate, American Boards of Environmental Medicine

Past President Capital University of Integrative Medicine

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Fatty Liver Reversal
Majid Ali, M.D.

Reversal With Insulin-Reducing Diet and Detox Program
Fatty liver is an unrecognized epidemic affecting all age groups. It is caused by the trio of toxicities of foods, environment, and thoughts. In most cases, this trio inflicts liver cell damage by triggering excess insulin production and creating a state of insulin toxicity. Both insulin toxicity and fatty change in the liver cause pathologic inflammation, which worsens the degrees of insulin-caused cell injury and fatty change.

What Is Fatty Liver?
Healthy liver cells are roughly rectangular in shape, have smooth surfaces and sharp cell boundaries, and contain centrally-located nuclei. Fatty liver is a state in which the cells are bloated with rancid fats. The nuclei are pushed to the periphery. The cell membranes are covered with grease composed of sticky sugars, rancid fats, and mangled proteins. Excess insulin pumps extra fats into the liver cells where they stagnate, become rancid, and literally suffocate and kill the liver cells. This is how fatty change in the liver develops in individuals with obesity, syndrome X, the metabolic syndrome, prediabetes, diabetes, atherosclerosis (hardening of arteries), hypertension, and inflammatory disorders.

Diagnosis of Fatty Liver
In the past, fatty liver was diagnosed by pathologists with microscopic studies of liver biopsies and liver tissues obtained during surgery and autopsy. Now advances in ultrasound technology make it possible to detect fatty change non-invasively. Since internists, gastroenterologists, and endocrinologists are not trained to detect and address the trio of toxicities of foods, environment, and thoughts, they ignore the twin problems of insulin toxicity and fatty liver. None of my patients with fatty change seen by these specialists had ever undergone a systematic insulin reduction program to reverse fatty change. I point out that all diabetes drugs increase insulin activity and increase the degree of fatty change.

My Top Priorities for Reversing Fatty Liver
Fatty change in the liver is a reversible lesion. If not reversed, it causes incremental liver cell death and leads to cirrhosis of the liver. Here are my top priorities for reversing fatty liver:

1. Self-compassion;
2. Bowel and liver detox in order to remove cellular grease and to normalize insulin receptor function;
3. Insulin-reducing diet (see fasting blood sugar)
4. Feather Breathing
5. Daily castor/sesame oil rubs
6. Lecithin and flaxseed as part of Dr. Ali's Breakfast
7. Hydrogen peroxide foot soaks
8. Limbic exercise (see www.ethicsinmedicine.us for details)
9. Liver-friendly spices and herbs. My most highly recommended items are Turmeric, milk thistle, dandelion, and black radish to be taken in weekly rotation (any two of the four can be rotated with the remaining two). Other options include goldenseal, red clover, Jerusalem artichoke, fennel seed, and catnip.

Reversal of Fatty Liver With Insulin-Reducing Program
A 67-year-old 5' 7" woman weighing 215 pounds consulted me for chronic fatigue syndrome, uncontrolled hypertension, gastric reflux disorder (GERD), obesity, allergy, and persistent leg edema. Previous doctors informed her of abnormal tests indicating inflammation in the liver of undetermined origin. Hepatitis tests were negative. Her health continued to deteriorate in spite of multiple drug therapies. Blood pressure remained high, sometimes as high as 240/120. On examination, her liver was tender to percussion (tapping). This finding, seen in light of abnormal liver tests (shown in Table 2), indicated significant inflammation and areas of cellular death in the liver. The 24-hour adrenal profile revealed a severe functional deficit. A toxic metal profile showed the following: lead, 13; arsenic, 12; mercury, 5.3; cadmium, 5.3 (all expressed as microgram /gram creatinine). Her four-hour insulin and glucose profiles are displayed in Table 1. These clinical and laboratory findings clearly establish the association of insulin toxicity and fatty change with liver cell death. The data in Table 2 also show dramatic reduction of inflammation in the liver with an insulin-reducing diet and detox program.

Table 1. The Insulin and Glucose Profile of a 67 year old 5 foot 7 inch 215 pound woman with Fatty Liver, Chronic Fatigue Syndrome, Hypertension, and Gastric Reflux Disorders 

Table 2. The two-hour postprandial insulin level fell from a high of 84.8 IU and 31.3 IU indicating a dramatic reduction of insulin load (and toxicity)

Other Causes of Fatty Liver
Alcohol-induced liver injury and drug toxicity are other major causes of fatty liver. Most drugs are broken down in the liver and cause liver injury. Notable among such drugs are amiodarone, methotrexate, diltiazem, antiretroviral agents, synthetic steroids, tamoxifen, and every chemotherapy drug. Environmental toxins of special concern are solvents, herbicides, pesticides, solvents, phosphorus, and mushroom poisoning. Other causes of fatty liver include: (1) severe weight loss; (2) malnutrition; (3) total parenteral nutrition; (4) stomach and bowel bypass operations; (5) colonic diverticulosis with bacterial overgrowth.

Insulin Toxicity
Insulin toxicity is the unrecognized spreading pandemic of our time. It has many clinical, microscopic, and bioenergetic faces, none of which is properly considered and addressed except by integrative clinicians. This is profoundly ironic for five reasons: (1) there is a vast body of literature on adverse effects of excess insulin (hyperinsulinemia); (2) inflammation plays crucial roles in nearly all chronic and subacute diseases, and insulin is a potent proinflammatory hormone; (3) insulin is obesitizing and obesity increases the risk of nearly all chronic and subacute diseases; (4) hyperinsulinemia is a strong risk factor of cardiovascular disorders; and (5) excess insulin plays central roles in the cause of metabolic disorders (such as polycystic ovary syndrome) and inflammatory disorders (fibromyalgia, vasculitis, and others).

Notable among the clinically significant and insufficiently appreciated faces of insulin toxcity are:
1. Fatty change of the liver,
2. Insulin-induced fat necrosis (a type of tissue death) in soft tissues,
3. Insulin dermatitis, including gray-yellow discoloration,
4. Endothelial dysfunction; and
5. Neuropathy.

Excess insulin is fattening and inflaming. Insulin toxicity turns a normal (physiological) healing inflammatory response to a disease-causing (pathologic) inflammation. Considered in this light, insulin toxicity fans the fires of all inflammatory disorders, whether involving the respiratory, cardiovascular, gastrointestinal, or other organ systems of the body. By controlling insulin toxicity, I have observed normalization of liver tests and reduction of PSA values in men with prostate cancer.